Case Report
ISSN: 2471 657X

Human Beings’ Sterile Compartments Osteon, Dental Implant or Odonton: If dead, out of the Head (The Case for Segmentectomy)

R.S. Carlson*
Inventor of the Carlson Bridge®..., USA
Corresponding author: R.S. Carlson,
inventor of the Carlson Bridge®..., USA. E-mail: ddscarlson@hawaiiantel.net
Received Date: June 13, 2018 Accepted Date: June 23, 2018 Published Date: July 15, 2018
Citation: R.S. Carlson. Human Beings’ Sterile Compartments Osteon, Dental Implant Or Odonton: If Dead, Out Of The Head (The Case for Segmentectomy). Int J Dent & Oral Heal. 4:7, 94-104.
Copyright: © R.S. Carlson. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited


 




Principle of Cleansing


This presentation keys off the biological Principle of Clea ing—sterility or purity. As we move through this document together we’ll return to this fundamental Principle clarifying why biological systems Cleanse, maintaining correct pure form and function—The True Principle Of Health. A key factor in this discussion is how we as dental surgical professionals may be aiding the natural process of Cleansing, and thus total human health, or obstructing it. We begin with the physiology of sterility of compartments of the human body, the starting point for our discussion. Normally “sterile sites” are defined and known as tissues such as: blood, lymphatic system, cerebrosp nal fluid (CSF), pleural fluid—chest fluid; peritoneal fluid—a dominal fluid, pericardial fluid; bone—bone marrow; joint fluid—synovial fluid, temporal mandibular joint, knee, ankle, elbow, hip, wrist; internal body sites, lymph node, brain, heart, liver, spleen, vitreous fluid, kidney, pancreas, ovary, vasculartissue, and most important for our discussion—The Odonton, a single functional unit of the masticatory system.

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Figure 1: Human Being sterile water content varies in different body organs and tissues, from as little as 8 percent in the teeth to as much as 85 percent in the brain

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Figure 1a:The Sterile Lower Molar Odonton

The Odonton is a sterile, pure, functional unit of the jaw until it is breached by micro-organisms.(1) Like the functional unit of bone, theOsteon, micro-organisms (m-o) do not play a role in normal biological tissue metabolism and function. It is accepted that “m-o” invasion of tissues, called “infection,” do the opposite, disrupt metabolism—form and function.

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Figure 2: Sterile Long Bone Osteon

Misdirections Of Dental Surgeons


For about 200 years, or more, there have been attempts to retain teeth whose sterile nutrient canals have been destroyed, compromised by putrefying m-os (microorganisms) by placing gutta-percha, sliver points, N2 Sargenti fillers, or other concoctions within the canals hoping for a return to sterility of the Odonton1 Thecontinuous contentions of the pro-root canal proponents and the so-called “Extractionest,” anti-root canal contingent, came to a head in the early 20th Century, well documented in the Dental Cosmos—monthly record of dental science 1859—1936. Case reports in the Dental Cosmos on dental causes of and systemic improvement of diseased conditions were actively reported such as this on “Systemic Conditions Due to Oral Focal Infections” cited here: https://quod.lib.umich.edu/d/dencos/0527912.0062.001/113:14?rgn=full+text;view=imag e and, here regarding “Focal Infections and Its Difficult Diagnosis” : https://quod.lib.umich.edu/d/dencos/0527912.0063.001/421:168?rgn=full+text;view=image;q1=oral+sepsis I suggest the readers review these discussions and get a sense of the intensity of the support for the Focal Theory and its controversy. As the years moved along discussions were vast and heated regarding focal dental infections and their systemic impact until about the 1945 when the Focal Theory Of Infection was allegedly discredited, not being taught in formal dental settings from then until today. However, as of this writing we see the reemergence of the concept, well accepted by general medicine, taking on a new name as Oral Systemic Infectious Connections. Interesting to note and curious is the fact that about this time of 1945 antibiotics, anti-inflammatory drugs and pain relievers were developed that could suppress symptoms of putrefaction and its gas production within the Odonton giving the false impression of cure.

Natural Essential Sterile Compartments


Revisiting the purity (sterility) compartments of the body, we note that of the lymphatic system, cerebrospinal fluid system, establish the blood brain barrier of the human brain compartment which are all designed in maintaining the sterility of the human brain compartment. Without which this sterility, the brain is subject to sever breakdown and death. The functioning unit of the human being is the brain, analogous to the functioning unit of an Odonton or Osteon. Metabolism takes place in a sterile environment for multiple reasons. The three main purposes of metabolism are the conversion of food and fuel to building structures for proteins, lipids, nucleic acids and some carbohydrates, and elimination of nitrogenous waste products. Enzymes of metabolism catalyze essential reactions in Life without the input of foreign items such as bacteria, viruses, prions, or other foreign microorganisms (m-os) ensuring a “pure pathways to successful metabolism”—growth and development. Our human design is impeccable in its purity, functions, and outcomes avoiding foreign input from exogenous or endogenous sources not overseen by innate predetermined DNA/RNA patterns. Common truthful sense will verify this, if we’ve take chemistry.

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Figure 3: Sterile Brain Compartment

Meningitis of the brain compartment linings is an inflammation of the membranes that surround the brain and spinal cord—violation of sterility. Most commonly caused by infections with various bacteria of viruses it is most terminal, if it can be treated at all. When the meninges are inflamed, the blood–brain barrier is disrupted. This disruption may increase the penetration of various substances into the brain. Antibiotics used to treat meningitis may aggravate the inflammatory response of the central nervous system by releasing neurotoxins from the cell walls of bacteria. The rare brain abscess is caused by inflammation and collection of lymphatic cells and infected material originating from a local or remote infection. A brain abscess is a rare, life-threatening condition. Local sources may include infections of the ear, the oral cavity and teeth, the Para nasal sinuses, or an epidural abscess. Remote sources may include infections in the lung, heart or kidney. A brain abscess may also be caused by head trauma or as a complication of surgery. In children cerebral abscesses are usually linked to congenital heart disease.

The author’s position is neither pro nor con regarding modern root canal therapy but bases this presentation on the fact that “sterility” is a True Principle Of Health ensured, empowerd by the Principle of Cleansing. Recall the fact that the functional unity of mastication, the Odonton, is a sterile space within many sterile spaces of the human body. The Odonton once dead—unsterile—now becomes a reservoir septic tank, nesting site, foci of infecting “m-os” that are pumped into the blood and lymph (sterile spaces) through masticatory action, pumping of the teeth. With proper observation of the “dead odontons” in Figure 4. bicuspid and Figure5. lower molar, we see the black, yellowish brown roots with soft apical abscess materials. Upon removal of impure root canal teeth we are struck with not only the color of the organ but also the odor often of putrefaction.

“What We’ve Called Modern Endodontic” For Centuries


Numerous innovative mechanical techniques have been developed over the years to render the canals sterile and uninhabitable for microorganisms. Virtually all educational courses and advertisements within professional journals emphasize “mechanical obturation” and not the biological aspects of sterility of the canal system, or if the canals can be sterilized at all in the short or long term. As reported recently, overemphasis on this aspect of simply filling the empty spaces within the root canal system and its importance has misled the field of endodontology.2 What is most critical is that root canal teeth become nesting sites, foci, for microbes and their “endotoxins” which, due to the loss of outward hydraulic pressure from within the body of the dental organ (since the pulp is necrotic, no circulation or oxygen within), migrate though the dentin into the peri-radicular tissues, thence into the systemic circulatory system and other tissues. In Dental Clinics of North America, Vol. 18, No. 2, April 1974, Kaare Langeland, D.D.S., Ph.D. reviewed “Root Canal Sealants and Pastes” in which he states:

The fact that sealers and pastes will contact vital or disintegrating tissue connected with the general circulation allows for generalized distribution by blood and lymph…The resorbabilty of sealers and pastes has been abundantly demonstrated by gross radiographic evidence. There is, therefore, no doubt that material is transported away from the site where it was originally applied”.

What has been shown today as of equal or more significant importance is the “sterility”—lack thereof—of the intra-radicular-canal system.[1,3,4,5] Current research shows that the significant presence of bacteria in the canal systems lead to the pathogenesis of “apical periodontitis” (AP)—now known as “endodontic disease.” This condition (AP) is a chronic/acute inflammation of the tissue about the roots of root filled teeth also designated as “apical granuloma.” [6,7]

Bacterial Action On Dead Tissues


Putrefaction, which is an essential stage in Odonton bio-film pulpal decomposition, is associated with a marked shift from aerobic bacterial species, which require oxygen to grow, to anaerobic ones, which do not. These anaerobic bacteria, called decomposers, subsist in a tenacious bio-film (plaque) attached to the various anatomical features of the root canal system, feeding on the degenerating pulp tissues, dentinal lymph fluids and their organic contents, other biological fluids from the dental crown andsurrounding periodontal ligaments and bone, thus fermenting the sugars in them to produce gaseous by-products such as methane, hydrogen sulphide and ammonia, and end products of impurities identified as cadaverene and putracine, which accumulate within the Odonton, inflating (or ‘bloating’) the pulp and root chambers through gas expansion—causing the “leaky root syndrome,” we call a tooth ache and pus-emission. These decomposition products noted previously cause further discoloration of the body of the Odonton. As damaged blood cells continue to leak from disintegrating vessels, anaerobic microorganisms convert haemoglobin molecules, which once carried oxygen around the body, into sulfhaemoglobin. The presence of this molecule in settled blood within a decomposing Odonton gives a greenish-black appearance, characteristic of a body undergoing active decomposition.

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Figure 4,5: lower bicuspid ,lower molar

Irrespective of the truth regarding purity or sterility of tissue spaces being rendered impure in the process of root canal therapy, it is still the only medical treatment advocating the active mummification or embalmment of human tissues, leaving it in the sterile human body. The orthopedic surgeon met with a necrotic Osteon (dead bone) in the advanced decay of the foot of a diabetic will resect the dead bone of the extremity.

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Figure 6: Impure Gangrene Of Toe Osteons

We present the truthful facts that when the human tissues are irretrievable damaged, necrotic, the first charge to the surgeon is to protect the purity of the other © Carlson Bridge Technologies, Inc. June 7, 2018 7 sterile compartments of the human body. In our discussion to this point we keep in view the vast number of pure/sterile compartments mentioned at the start of this presentation, paragraph two. As with Osteons of the toes Odontons of the mouth should be resected too. In Figure 6 it would be critical to the general health of the human to remove the gangrene tissue of the toes with intent to maintain purity of the lymph system, remaining bones of the foot, ligaments, muscles and blood system. The end result of not doing this would be to the jeopardy of the Life of the individual. In the oral cavity we have many instances of Odonton’s appearing as the toes in Figure 6 but not so black or dramatic until removed. Here is one in Figure 7 demonstrating a “gum boil” or a parulus extruding pus on the surface, right of crown.

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Figure 7: Gold Crown Over Necrotic Odonton #19

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Figure 8: X-Ray Root Canal Odonton #19

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Figure 9: Necrotic Root Canal #19

Once the necrotic root canal #19 was removed we see the soft tissue below the root tips but intimately attached thereto, representing the very unsterile condition of the Odonton and the bone associated. We for 35 years have submitted theses tissues to a board certified pathologist at Queens Hospital Pathology Laboratory and offer the findings in this specific individual:

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Figure 10:

In over 300 reports for the past 35 years we find similar results as reported in this one, “…marked acute/chronic inflammation, necrotic bone, reactive bone, osteomyelitis, cystic granulomata, with notations in about 50% of the reports of filamentous bacteria consistent with Actinomyces.”

Lifeless Impure Dental Implants


Periodontal Disease pre se, Apical Periodontitis (endodontic disease), Peri- Implant Mucosities, and Peri-Implantitis pathogens—all basically the same, namely Actinomyces odontolyticus et al—have been linked with increased risk of systemic illness and complications in existing disease.[8,9] Many articles detailing these findings have been published emphasizing the importance of the association between oral conditions as noted above and systemic health problems. In fact, a recent meta-analysis reports that oral infections have been shown to be associated statistically with mortality. By entering the bloodstream, periodontal pathogens have been shown to increase the risk of cardiovascular and pulmonary disease, and hinder glycemic control in diabetes. Due to the fact that the “micro-gap” at the dental implant junction and the gum/bone are NONCLEANSABLE (Impure), dental implants may inoculate “mo-s” into the bone interface of the implant through micro-movements, pumping action, deep in the gum-bone support tissue gap, so that bacteria and their toxins are thrust into the sterile blood and lymph tissues spaces—increasing risks to systemic health.[8,9,10]

In the photo of Figure 11 one observes a dental implant (to the right of the Gold Crown) demonstrating the loss of bone with pus extruding from the “micro-gap.” The xray in Figure 12 reveals the cupping of crest bone around the implant #29 and at its apex (arrows), the radiolucent evidence of abscess. Figure 13 shows the deep abscess material about the dental implant #29 and in Figure 14 we show the histopathology of the condition that was asymptomatic other than a general systemic malaise. There was necrotic bone, Actinomyces, Granuloma, and chronic inflammation.

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Figure 11: Dental Implant Pus

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Figure 12: Post- X-Ray #29 Dental Implant

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Figure 13: Post-Surgical Metal Implant And Pathological Tissues

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Figure 14: Pathologic Report Metal Implant #29

Actinomyces And Actinomycosis


Biofilms harboring the anaerobic bacterial species Actinomyces are the new challenges to be overcome by “modern endodontics” in the spaces that were once pure, the sterile dental organ, Odonton.[1] In the early 1980s we began to remove infected root canal teeth and associated soft tissue and bone (Odonton) and send them for pathological inspection by a board certified histopathologist. The findings were remarkably revealing to this date. In brief all of the 349 “odontons” inspected it was found that acute-chronic inflammation—sometimes marked, reactive bone, dead bone, osteomyelitis, granuloma, cyst, abscess and other attending abnormalities were present in the jaw below the tooth in question. Having carried this root canal research on since about 1983, we have also extended this methodology to implant removal due to infections or otherwise.

Background on factors causing “marked acute-chronic inflammation.” As we know the dead tooth has no circulatory system internally—in a sense this is the definitionof an endodontically treated tooth. As the dead tooth degrades it gives off variouschemicals such as putresciene, cadaverine, thioethers, and what we call “endo-toxins.”This fact is no longer disputed in the scientific clinical profession, nor in academia.Another factor for inflammation is bacterial presence in tissues where they shouldnot be—sterile compartments throughout the human body. As a habit in the early days ofour research the pathologist noted what he called “star-like microorganisms” in colonieswith rays in the bone and soft tissues. He noted that they were certainly colonies ofactinomyces that surrounded the root canal. However, it must be understood that we hadnot asked for a determination of bacterial review, it was an incidental finding in hispathology reports.

Historically, Actinomyces in know from the 1800s as the “ray fungus,” mistakenas a fungus until about 1938. It is currently identified as a bacterium: Actinomyces isa genus of the Actinobacteria class of bacteria—all being grampositive. Actinomyces species are facultatively anaerobic and they grow best underanaerobic conditions. Actinomyces species may form endospores, and, while individualbacteria are rod-shaped, Actinomyces colonies form fungus-like branched networksof hyphae that may transform into other shapes and actions.[10]

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Figure 15: Microorganism And Their Morphological Changes

Gram-stained of Candida. albicans cultures observed under light microscopy at 1000× magnification. Left: Candida albicans(ALT4) grown in RPMI-1640 after 24 h incubation at 37°C; >75% ofCandida albicans cells were present in hyphal form in this medium. Right: Candida albicans (ALT4) grown inASM after 24 h incubation at 37°C; 100% of Candida albicans displaying yeast morphology in this medium.

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Figure 16: Actinomyces

Microorganisms transmute, change form, very often depending on biofilms available; and, as co inhabitants auto aggregating or co aggregating. Morphologicalvariations are normal and misidentifications arise as a rule of order in this field.Candida and Actinomyces have been implicated in decay and gum or periodontaldisease in the oral cavity since they are normal residents. However, it is not normal to find either candida/actinomyces in bone or connective tissues deep in the other jawboneor in the blood vascular system. If found in the jaw bone it may lead to a condition known as “lumpy jaw”—an abscess from either dental or peri-dental origins, or both. Systemically it may lead to Actinomycosis a condition where abscesses occur—through circulatory migration or facial migration—into jaw, throat, lungs, abdomen, genital areas.

Actinobacteria are the most common cause of infection in dental procedures and deep state oral abscesses related to apical periodontitis—root canals. Many Actinomyces species are opportunistic pathogens of humans and other mammals, particularly in the oral cavity. In rare cases, these bacteria can cause actinomycosis, a disease characterized by the formation of abscesses in the mouth, lungs, or the gastrointestinal tract. Actinomycosis is most frequently caused by A. isralli, which may also cause endocardidis, though the resulting symptoms may be similar to those resulting from infections by other bacterial species. Aggreatibacter actinomycetemcomitans has been identified as being of note in periodontal disease. The genus is typically the cause oral-cervicofacial disease. Lymphadenopathy is uncommon in this form of this condition. Another form of actinomycosis is thoracic disorder, which is often misdiagnosed as a neoplasm, as it forms a mass that extends to the chest wall. It arises from aspiration of organisms from the oropharynx. Symptoms include chest pain, fever, and weight loss. Abdominal disturbances are another manifestation of actinomycosis. This can lead to a sinus tract that drains to the abdominal wall or the perianal area. Symptoms include fever, abdominal pain, and weight loss. Actinomyces species have also been shown to infect the nervous system without apparent trauma and could be related to Lyme disorder that is now the recognized malady of the day.

The bacteria Actinomyces are ubiquitous and abundant in soil. They are known for the important role they play in soil ecology; they produce a number of enzymes that help degrade plant root debris and waste plant materials as do Spirochetes and Candida. A specific Spirochete has been implicated as the source of Lyme but Actinomyces may be associated likewise. Thus their presence is important in the formation of compost, as are spirochetes and candida fungi, the later specialist in degradation of cellulose. They are also known for causing diseases in humans and livestock, usually when they get an opportunity to gain access to the body’s interior through wounds. As with other opportunistic infections, people with immunologic issues are at higher risk. In all of the preceding traits and in their branching filament formation, they bear similarities to Norcardia causing Norcardosis, a similar illness as Actinomycosis.

It is well established that actinomycosis is an endogenous infection. The causative Actinomyces species reside on mucosal surfaces and gain access to deeper tissues via trauma, surgical procedures, or foreign bodies, which disrupt the mucosal © Carlson Bridge Technologies, Inc. June 7, 2018 14 barrier. Inside the tissue these bacteria form masses consisting of aggregates of branching, filamentous bacilli. Actinomycosis is defined as a hard mass-type lesion with a specific histopathological structure. There are a large number of case reports of actinomycosis in the literature, but in most cases, diagnosis has been based solely on clinical and histopathological findings. In the majority of early reports, microbiological confirmation of diagnosis was lacking and confusing. Even when microbiological assessment was included, culture was typically the only method used. If, however, antimicrobial treatment had been started before sample collection, the results of culture may be falsely negative. The increasing introduction of molecular bacterial detection and identification methods is helping to overcome such problems.

An internal abscess is more difficult to identify, but signs include pain in the affected area, a high temperature, and generally feeling unwell. Internal abscesses rarely heal themselves, so prompt medical attention is indicated if such an abscess is suspected.This is important in the resolution of infected abscessed teeth, that they be surgically removed and bone cleansing—cavitation—accomplished.

Closing Thoughts


This paper discusses the sterile physiological spaces making up the human being andtheir relationship to THE PRINCIPLE OF HEALTH and the PRINCIPLE OF CLEANSING. If one chooses to offer “root canals,” one is locked into thepharmacological trap of combative chemistry and surgeries forestalling the inevitable—segmentectomy. Our “Opioid Crisis” today may be directly related to optional medicalprocedures blocking complete remission to adverse medical treatments. See here:https://www.democracynow.org/2018/6/1/origins_of_the_opioid_epidemic_purdueThe methodologies in this day and age lead to the oral-systemic toxemia that very innocuously infects other pure/sterile spaces of the human body. Resurgence of the ideathat the human body is a “totality” and not series of parts unconnected leading toimprovement or demise is now manifesting in research literature and in the awareness ofthe general population. The knowledge that pure spaces are to be maintained as purespaces is verified in my experience in the return to health in individuals choosing to remove infected matter from the oral cavity—root canals, dental implants, periodontally involved Odontons.

As noted earlier the author’s position is neither pro nor con regarding the socalled “modern root canal therapy” but bases this presentation on the truthful fact that“sterility” is a True Principle Of Health empowered by the Principle of Cleansing. Recall the fact that the functional unity of mastication, the Odonton, is a sterile space within many sterile spaces of the human body. The Odonton once dead—unsterile—now becomes a reservoir septic tank, nesting site, foci of infecting m-os that are pumped into the blood and lymph (sterile spaces) through masticatory action, pumping of the teeth. In the final analysis it is the choice of the attending dental surgeon and his or her patient that will make the difference in patient’s health. Choices will always be based upon “pro or con” in the context of scientific debate on the subject matter; but, if one © Carlson Bridge Technologies, Inc. June 7, 2018 15 simply looks at the “sterile/pure space” issue, that is “sterile compartments,” The True Principle Of Health, the Principle Of Cleansing as fundamentals of health in the human, one could not tolerate soiling the human body, thus avoiding being an obstruction to health.


References:

  1. Figdor, et al, Survival against the odds: microbiology of root canals associated with post-treatment disease. Endodontic Topics 2011, 18, 62-77.
  2. Figdor, David. Guest Editorial Apical periodontitis: a very prevalent problem; Oral Surgery, Oral Medicine, Oral Pathology, Oral Radiology & Endodontics, Vol. 94 No. 6, December 2002.
  3. Ibid.
  4. Sjorgen U, Figdor D. Persson S. Sundquist G. Influence of infection at the time of root filling on the outcome of endodontic treatment of teeth with apical periodontitis. Int Endod J 1997; 30:297-306.
  5. Katebzadeh N, Hupp J, Trope M. Histological periapical repair after obturation of infected root canals in dogs. J Endod 1999; 25: 364-8.
  6. Katebzadeh N, Sigurdsson A, Troope M. Radiographic evaluation of periapical healing after obturation of infected root canals: an in vivo study. Int Endod J 2003:33:60-6.
  7. Regezi, J. Periapical Diseases: Spectrum and Differentiating Features; Journal of the California Dental Association, April 1999.
  8. Mohammad Shahabouee,1 Mansour Rismanchian,2 Jaber Yaghini,1 Akram Babashahi,3Hamid Badrian,3 and Hossein Goroohi3; Microflora around teeth and dental implants; Dent Res J (Isfahan). 2012 Mar-Apr; 9(2): 215–220.
  9. Rafał Pokrowiecki,1 Agnieszka Mielczarek,2 Tomasz Zaręba,3 and Stefan Tyski3,4; Oral microbiome and peri-implant diseases: where are we now?; Ther Clin Risk Manag. 2017; 13: 1529–1542.
  10. Mohd Hafiz Arzmi Stuart Dashper Deanne Catmull Nicola Cirillo Eric C. Reynolds Michael McCullough; Coaggregation of Candida albicans, Actinomyces naeslundii and Streptococcus mutans is Candida albicans strain dependent; FEMS Yeast Research, Volume 15, Issue 5, 1 August 2015.

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